This product is free for those who attended FNCE® 2016.
Individuals with type 2 diabetes are characterized by prolonged periods of hyperglycemia due to a decreased responsiveness of skeletal muscle to the actions of insulin (insulin resistance). A primary goal for the treatment of type 2 diabetes should be to enhance skeletal muscle oxidative capacity to improve metabolic flexibility (i.e., the ability to transition between carbohydrate- and fat-based fuels), and improve post-prandial glycemic control.
This presentation will review the mechanisms of action as well as practical applications by which novel dietary and exercise interventions can improve whole-body glucose homeostasis by modulating skeletal muscle metabolic flexibility.
CPE Level: 3
Learning Codes: 5190, 2110, 4040
Planned with the Academy's Committee for Lifelong Learning